The relationship between the thyroid-related hormone T3 and hypothyroidism has been known for some time. Simply stated, a lack of T3 will often impact how the thyroid functions. This may lead to a host of symptoms, such as lethargy, memory loss, decreased libido, and weight gain. However, it is also a well-established fact that science continues to make astounding advancements. Let’s take a look at some new discoveries related to hypothyroidism, as well as why hormone replacement still represents one of the most viable forms of therapeutic treatment.
T3 and Thyroxine
Although many of us might already be aware of T3, relatively few appreciate the role that T4 plays. T4 is also known as thyroxine, and it is formed within the thyroid. Assuming that this gland is functioning normally, T4 will be sent to the liver via the bloodstream. It is thereafter converted into T3. If this conversion is unable to take place (such as in the event that not enough T4 is being produced), many of the metabolic effects mentioned in the previous section may come to pass. In other words, scientists have a much better understanding of the synergistic relationship between these two essential hormones.
Might Genetics Play a Greater Role Than Suspected?
One of the most potent ways to counteract the effects of hypothyroidism involves the administration of quality supplements such as the T3 tablets online here. Having said this, researchers are also beginning to uncover a link between genetics and the likelihood of developing thyroid disorders. What have they found?
Although this is a highly technical subject, it can still be summarised from a broad perspective. There are certain enzymes within the body known as deiodinases. Deiodinases are primarily responsible for converting T4 into T3. At times, specific mutations can cause these deiodinases to function improperly. These are known as “polymorphisms”. It is thought that some individuals may be more likely than others to develop these types of mutations. As a result, they could also be prone to thyroid issues.
This leads us to an important question. Might it be possible to develop some type of genetic testing in order to determine one’s potential predisposition? If so, could this lead to more targeted treatment options and the ability to tackle the symptoms associated with hypothyroidism at an earlier stage? Considering how much genetic research has progressed in recent times, these concepts could soon become realities.
The main takeaway point here is that there are plenty of ways in which you can mitigate the effects of an underactive thyroid gland. These primarily include
taking specific medications and embracing a handful of healthy lifestyle changes. This is also the reason why many individuals who have already been diagnosed with hypothyroidism can now live their lives to the fullest.
Might there come a day when a cure for thyroid conditions is discovered? Although the verdict is still out, there is no doubt that advancements will continue to streamline clinical approaches well into the future.
